Researcher Database

NOMA Akinori

Department / Course Research Organization of Science and Technology

Title / Position Visiting Senior Researcher

Papers
1.	2018/09	A new myofilament contraction model with ATP consumption for ventricular cell model │ The Journal of Physiological Sciences │ 68 (5),pp.541-554 (Co-authored)
2.	2017/04	Regulation of the glucose supply from capillary to tissue examined by developing a capillary model │ J Physiol Sci. │ 68 (4),pp.355-367 (Co-authored)
3.	2016/01	Mechanisms underlying the volume regulation of the interstitial fluid by capillaries: a simulation study │ Integrative Medicine Research │ 5 (1),pp.11-21 (Co-authored)
4.	2015/11	Modeling analysis of inositol 1,4,5-trisphosphate receptor-mediated Ca mobilization under the control of glucagon-like peptide-1 in mouse pancreatic b-cells. │ Am J Physiol Cell Physiol │ 310 (5),pp.C337-47 (Co-authored)
5.	2015/07	A human ventricular myocyte model with refined representation of excitation-contraction coupling │ Biophys. J │ 109 (2),pp.415-427 (Co-authored)
6.	2014/09	EAD and DAD mechanisms analyzed by developing a new human ventricular cell model │ Progress in Biophysics and Molecular Biology │ 116 (1),pp.11-24 (Co-authored)
7.	2012	Steady-state solutions of cell volume in a cardiac myocyte model elaborated for membrane excitation, ion homeostasis and Ca2+ dynamics. │ J Theor Biol │ 307,pp.70-81 (Co-authored)
8.	2011/11	Analyzing electrical activities of pancreatic β cells using mathematical models, │ Prog Biophys Mol Biol. │ 107 (2),pp.265-73 (Co-authored)
9.	2011/07	Ionic mechanisms and Ca2+ dynamics underlying the glucose response of pancreatic β cells: a simulation study, │ J Gen Physiol │ 138 (1),pp.21-37 (Co-authored)
10.	2011/07	Time-dependent changes in membrane excitability during glucose-induced bursting activity in pancreatic β cells, │ J Gen Physiol │ 138 (1),pp.39-47 (Co-authored)
11.	2011/04	Reply to "Letter to the Editor: 'Validating the requirement for beat-to-beat coupling of the Ca2+ clock and M clock in pacemaker cell normal automaticity' │ Am J Physiol Heart Circ Physiol │ 300 (6),pp.H2325-H2326 (Co-authored)
12.	2011	Minor Contribution of Cytosolic Ca2+ Transients to the PacemakerRhythm in Guinea Pig Sinoatrial Node Cells. │ Am J Physiol Heart Circ Physiol, │ 300 (1),pp.H251-61 (Co-authored)
13.	2011	Systems Analysis of GLP-1 Receptor Signaling in Pancreatic β-cells. │ Am J Physiol Cell Physiol. │ 138 (1),pp.39-47 (Co-authored)
14.	2010/10	A, Minor contribution of cytosolic Ca2+ transients to the pacemaker rhythm in guinea pig sinoatrial node cells, │ Am J Physiol Heart Circ Physiol. │ 300,pp.H251-61 (Co-authored)
15.	2010	Characterization of the cardiac Na+/K+ pump by development of a comprehensive and mechanistic model │ J Theor Biol │ 256 (1),pp.68-77 (Co-authored)
16.	2010	Characterization of the cardiac Na+/K+ pump by development of a comprehensive and mechanistic model │ J Theor Biol │ 265 (1),pp.68-77 (Co-authored)
17.	2010	Magnesium gating of cardiac gap junction channels │ Prog Biophys Mol Biol │ 103 (1),pp.102-110 (Co-authored)
18.	2009	A Novel Method to Quantify Contribution of Channels and Transporters to Membrane Potential Dynamics │ Biophys J. │ 97 (12),pp.3086-94 (Co-authored)
19.	2009	A Novel Method to Quantify Contribution of Channels and Transporters to Membrane Potential Dynamics, │ Biophys J. │ (Co-authored)
20.	2009	Development of a model of the Na+/H+ exchanger and its central role in regulation of intracellular pH and Na+ in cardiac myocytes │ Biophys J. │ 97 (10),pp.2674-83 (Co-authored)
21.	2009	Development of a model of the Na+/H+ exchanger and its central role in regulation of intracellular pH and Na+ in cardiac myocytes, │ Biophys J. │ (Co-authored)
22.	2009	Modeling the cardiac Na+/H+ exchanger based on major experimental findings, │ Mol Cells. │ (Co-authored)
23.	2009	Modeling the cardiac Na+/H+ exchanger based on major experimental findings, Mol Cells. │ Mol Cells. │ 28 ((2),),pp.p81-5 (Co-authored)
24.	2008	A new integrated method for analyzing heart mechanics using a cell-hemodynamics -autonomic nerve control coupled model of the cardiovascular system │ Prog Biophys Mol Biol │ 96 (1-3),pp.44-59 (Co-authored)
25.	2008	A simulation study on the activation of cardiac CaMKII delta-isoform and its regulation by phosphatases │ Biophys J │ 95,pp.2139-49 (Co-authored)
26.	2008	A simulation study on the activation of cardiac CaMKII delta-isoform and its regulation by phosphatases │ Biophys J │ 95,pp.2139-49 (Co-authored)
27.	2008	Ionic mechanisms underlying the positive chronotropy induced by beta1-adrenergic stimulation in guinea pig sinoatrial node cells: s simulation study │ J Physiol Sci │ 58,pp.53-65 (Co-authored)
28.	2008	Ionic mechanisms underlying the positive chronotropy induced by beta1-adrenergic stimulation in guinea pig sinoatrial node cells: s simulation study │ J Physiol Sci │ 58,pp.53-65 (Co-authored)
29.	2007	Ionic mechanisms of cardiac cell swelling induced by blocking Na+/K+ pump as revealed by experiments and simulation │ J Gen Physiol │ 128,pp.495-507 (Co-authored)
30.	2007	Ionic mechanisms of cardiac cell swelling induced by blocking Na+/K+ pump as revealed by experiments and simulation │ J Gen Physiol │ 128,pp.495-507 (Co-authored)
31.	2007	Resistance of Cardiac Cells to NCX Knockout: A Model Study │ Annals of the New York Academy of Sciences │ 1099,pp.306-309 (Co-authored)
32.	2007	Resistance of Cardiac Cells to NCX Knockout: A Model Study │ Annals of the New York Academy of Sciences │ 1099,pp.306-309 (Co-authored)
33.	2007	Role of Ca2+ transporters and channels in the cardiac cell volume regulation │ Annals of the New York Academy of Sciences │ 1099,pp.377-382 (Co-authored)
34.	2007	Role of Ca2+ transporters and channels in the cardiac cell volume regulation │ Annals of the New York Academy of Sciences │ 1099,pp.377-382 (Co-authored)
35.	2007	Simulation analysis of intracellular Na+ and Cl- homeostasis during beta 1-adrenergic stimulation of cardiac myocyte │ Prog.Biophys Mol Biol │ 96,pp.171-186 (Co-authored)
36.	2007	Simulation analysis of intracellular Na+ and Cl- homeostasis during beta 1-adrenergic stimulation of cardiac myocyte │ Prog.Biophys Mol Biol │ 96,pp.171-186 (Co-authored)
37.	2007	The Cross-Bridge Dynamics during Ventricular Contraction Predicted by Coupling the Cardiac Cell Model with a Circulation Model │ J Physiol Sci │ 57,pp.275-285 (Co-authored)
38.	2007	The Cross-Bridge Dynamics during Ventricular Contraction Predicted by Coupling the Cardiac Cell Model with a Circulation Model │ J Physiol Sci │ 57,pp.275-285 (Co-authored)
39.	2006	A Simulation Study to Rescue the Na+/Ca2+ Exchanger Knockout Mice │ The Journal of Physiological Sciences │ 56,pp.211-217 (Co-authored)
40.	2006	Calcium-mediated coupling between mitochondrial substrate dehydrogenation and cardiac workload in single guinea-pig ventricular myocytes │ Journal of Molecular and Cellular Cardiology │ 40,pp.394-404 (Co-authored)
41.	2006	Modeling the Ca2+ gate on cardiac gap junction channel │ J.Physiol.Sci │ 56,pp.79-85 (Co-authored)
42.	2006	Modelling Cl- homeostasis and volume regulation of the cardiac cell │ Philos Transact A Math Phys Eng Sci │ 364,pp.1245-1265 (Co-authored)
43.	2006	simBio: A java package for the development of detailed cell models │ Prog Biophys Mol Biol │ 90,pp.360-377 (Co-authored)
44.	2006	The Cl- homeostasis and volume regulation of the cardiac cell model │ Philos Transact A Math. Phys. Eng. Sci │ 364 (1-3),pp.1245-1265 (Co-authored)
45.	2005	Regulation of oxidative phosphorylation in intact mammalian heart in vivo │ Biophysical Chemistry │ 116,pp.145-157 (Co-authored)
46.	2004	An in silico study of energy metabolism in cardiac excitation-contraction coupling │ Japanese Journal of Physiology │ 54,pp.517-522 (Co-authored)
47.	2004	Simulation of ATP Metabolism in Cardiac Excitation-Contraction Coupling │ Progress in Biophysics and Molecular Biology │ 85,pp.279-299 (Co-authored)
48.	2004	Two types of spontaneous depolarizations in the interstitial cells freshly prepared from the murine small intestine │ Journal of Physiology │ 599,pp.411-422 (Co-authored)
49.	2004	Two types of spontaneous depolarizations in the interstitial cells freshly prepared from the murine small intestine. │ J Physiol. │ 55,pp.411-422 (Co-authored)